American Journal of Respiratory Cell and Molecular Biology
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LPS induced acute lung injury involves the NF-?B-mediated downregulation of Sox18
Dimethylarginine dimethylaminohydrolase II overexpression attenuates LPS-mediated lung leak in acute lung injury.
Heat shock protein 90 inhibitors prevent LPS-induced endothelial barrier dysfunction by disrupting RhoA signaling.
Novel mechanism of attenuation of LPS-induced NF-?B activation by the heat shock protein 90 inhibitor, 17-N-allylamino-17-demethoxygeldanamycin, in human lung microvascular endothelial cells.
Protein kinase C-? and arginase I mediate pneumolysin-induced pulmonary endothelial hyperpermeability.
C-terminus of heat shock protein 70-interacting protein-dependent GTP cyclohydrolase I degradation in lambs with increased pulmonary blood flow.
Heat shock protein 90 inhibitors protect and restore pulmonary endothelial barrier function.
Estrogen reduces carbachol-induced constriction of asthmatic airways by stimulating large-conductance voltage and calcium-dependent potassium channels.